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. Author manuscript; available in PMC: 2020 Jul 31.
Published in final edited form as: Aliment Pharmacol Ther. 2019 Jun 5;50(2):121–131. doi: 10.1111/apt.15306

Table 2b.

H pylori exposure and CagA serostatus among individuals with CD compared to individuals without IBD

Author, Year CD No IBD CD No IBD
HP− HP+ HP− HP+ CagA− CagA+ CagA− CagA+
Lord, 2018a 194 18 223 34 209 2 235 19
Rosania, 2018b 73 17 138 42 87 3 161 19
Wagtmans, 1997c 339 47 179 98 355 31 209 68
a

This study included H. pylori (HP) positive, trace, and negative samples. No information was provided delineating positive from trace positive, but the study considered trace to be a “non-positive” result. Accordingly, we considered “trace” to be HP negative for this analysis. In addition, CagA samples were “missing” for 6 samples total (1 in CD, 2 in UC, and 3 in controls).

b

Based on data presented in this study, the HP positive subjects in the UC and CD subgroups do not add up to the total HP positive subjects in the IBD group and the reason for the discrepancy is not provided by the authors.

c

This study only included patients with CD and not UC