. Author manuscript; available in PMC: 2020 Jul 31.

Published in final edited form as: Aliment Pharmacol Ther. 2019 Jun 5;50(2):121–131. doi: 10.1111/apt.15306

Table 2c.

H pylori exposure and CagA serostatus among individuals with UC compared to individuals without IBD

Author, Year	UC		No IBD		UC		No IBD
Author, Year	HP−	HP+	HP−	HP+	CagA−	CagA+	CagA−	CagA+
Lord, 2018^a	211	24	223	34	221	12	235	19
Rosania, 2018^b	32	5	54	20	34	3	66	8
Wagtmans, 1997^c

This study included H. pylori (HP) positive, trace, and negative samples. No information was provided delineating positive from trace positive, but the study considered trace to be a “non-positive” result. Accordingly, we considered “trace” to be HP negative for this analysis. In addition, CagA samples were “missing” for 6 samples total (1 in CD, 2 in UC, and 3 in controls).

Based on data presented in this study, the HP positive subjects in the UC and CD subgroups do not add up to the total HP positive subjects in the IBD group and the reason for the discrepancy is not provided by the authors.

This study only included patients with CD and not UC