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. 2020 Jul 30;19:1533033820945799. doi: 10.1177/1533033820945799

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© The Author(s) 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — MiR-182-5p is a novel downstream molecule of SFMBT2 in glioma. (A) The complementary binding sites between miR-182-5p and SFMBT2 transcript were predicted. (B) Overexpressed miR-182-5p lead to significantly decreased luciferase activity of SFMBT2-WT, but not in SFMBT2-MUT. (C and D) The level of miR-182-5p was notably elevated in glioma samples. (E and F) The expression of miR-182-5p in D54 and A172 cells was upregulated. *P < .05. MiR indicates microRNA; miR-182-5p, microRNA-182-5p; MUT, mutant; SFMBT2, circular RNA Scm like with 4 Mbt domains 2; WT, wild-type.