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. 2020 Jun 11;11(8):2209–2223. doi: 10.1111/1759-7714.13528

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© 2020 The Authors. Thoracic Cancer published by China Lung Oncology Group and John Wiley & Sons Australia, Ltd

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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mPRα mediates the inhibitory effect of P4/Org on lung adenocarcinoma cells. (a) The growth curves of PC‐9/PC‐9‐mPRα⁻ cells treated with 20 μM P4/Org for different times (24, 48, and 72 hours), as detected by a CCK‐8 assay () PC‐9, () PC‐9+P4, () PC‐9+Org, () PC‐9‐mPRα⁻, () PC‐9‐mPRα⁻+P4, () PC‐9+mPRα⁻+Org. (b) Effects of 20 μM P4/Org treatment on the formation of PC‐9 and PC‐9‐mPRα⁻ cell colonies. (c) Analysis of the results of colony formation experiments (n = 3) () Control, () P4, () Org. * P < 0.05; ** P < 0.01.

Inline graphic — mPRα mediates the inhibitory effect of P4/Org on lung adenocarcinoma cells. (a) The growth curves of PC‐9/PC‐9‐mPRα⁻ cells treated with 20 μM P4/Org for different times (24, 48, and 72 hours), as detected by a CCK‐8 assay () PC‐9, () PC‐9+P4, () PC‐9+Org, () PC‐9‐mPRα⁻, () PC‐9‐mPRα⁻+P4, () PC‐9+mPRα⁻+Org. (b) Effects of 20 μM P4/Org treatment on the formation of PC‐9 and PC‐9‐mPRα⁻ cell colonies. (c) Analysis of the results of colony formation experiments (n = 3) () Control, () P4, () Org. * P < 0.05; ** P < 0.01.