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. Author manuscript; available in PMC: 2020 Aug 3.
Published in final edited form as: J Pharm Biomed Anal. 2018 Jun 18;158:351–360. doi: 10.1016/j.jpba.2018.06.022

Table 3.

Kinetic parameters derived for NBIF-related glucuronides (G1 and G2) by HLM, HIM, expressed UGT enzymes, MkLM, RLM, MLM, DLM and RaLM (mean ± SD). All experiments were performed in triplicate.

Enzyme Meta. Vmax (pmol/min/mg) Km (μM) Ki (μM) CLint (μL/min/mg) Model
HLM G1 1788.0 ± 203.6 6.58 ± 1.25 48.45 ± 13.86 271.90 ± 60.39 SI
G2 919.1 ± 20.23 1.41 ± 0.13 N.A. 651.38 ± 60.31 MM
HIM G1 409.3 ± 18.9 6.83 ± 0.91 N.A. 59.96 ± 8.42 MM
G2 8655.0 ± 1021.0 9.12 ± 1.64 33.32 ± 7.95 949.01 ± 203.75 SI
UGT1A1 G1 912.4 ± 49.36 3.64 ± 0.48 291.1 ± 100.1 250.87 ± 35.80 SI
G2 48.33 ± 1.59 1.03 ± 0.11 401.2 ± 135.7 47.01 ± 5.35 SI
UGT1A7 G1 11.93 ± 0.55 0.41 ± 0.06 61.82 ± 13.5 28.87 ± 4.57 SI
G2 135.3 ± 4.36 0.31 ± 0.04 154.5 ± 44.85 438.15 ± 56.41 SI
UGT1A8 G1 419.1 ± 18.69 4.52 ± 0.54 N.A. 92.68 ± 11.73 MM
G2 147.0 ± 6.84 4.0 ± 0.51 N.A. 36.75 ± 5.00 MM
UGT1A9 G1 70.01 ± 1.37 1.77 ± 0.14 N.A. 39.49 ± 3.11 MM
G2 805.9 ± 17.46 0.75 ± 0.07 N.A. 1073.25 ± 108.93 MM
UGT2B4 G2 + + + +
MkLM G1 4129.0 ± 182.8 11.39 ± 1.25 N.A. 362.51 ± 42.87 MM
G2 7072.0 ± 228.4 5.62 ± 0.55 N.A. 1257.92 ± 129.47 MM
RLM G1 2490.0 ± 249.5 6.40 ± 1.01 24.73 ± 4.75 388.88 ± 72.83 SI
G2 826.6 ± 99.48 2.95 ± 0.57 9.09 ± 1.82 280.11 ± 63.39 SI
MLM G1 3589.0 ± 432.9 7.41 ± 1.43 38.19 ± 10.18 484.67 ± 110.41 SI
G2 3878.0 ± 563.1 8.84 ± 1.96 31.68 ± 9.16 438.64 ± 116.08 SI
DLM G1 1807.0 ± 48.14 9.31 ± 0.65 N.A. 194.09 ± 14.55 MM
G2 3363.0 ± 85.47 3.48 ± 0.30 N.A. 965.27 ± 86.90 MM
RaLM G1 3006.0 ± 103.8 4.82 ± 0.43 N.A. 623.26 ± 60.06 MM
G2 3411.0 ± 155.5 4.13 ± 0.51 N.A. 826.61 ± 109.47 MM

Note: Meta., metabolites; N.A., not available; +, under the limit of quantification; HLM, human liver microsomes; HIM, human intestine microsomes; MkLM, monkeys liver microsomes; RLM, rats liver microsomes; DLM, dogs liver microsomes; MLM, mice liver microsomes; RaLM, rabbits liver microsomes; SI, substrate inhibition model; MM, Michaelis-Menten model.