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. 2020 Jul 28;10:375. doi: 10.3389/fcimb.2020.00375

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Copyright © 2020 Hsieh, Tsai, Chen, Shen, Jhan, Tseng and Lin.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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A hypothetical model of enhanced DENV infection in senescent monocytes. In this study, the chemical D-galactose-induced senescent response in monocytic THP-1 cells caused IL-10 production through an unknown mechanism. Then, IL-10 triggered JAK2-STAT3 signaling-regulated DC-SIGN expression to enhance DENV infection.