Figure 3.

(a) Serum ALT levels according to the etiology of acute liver injury. The serum ALT level differed significantly according to the etiology of acute liver injury (p<0.0001). In the infectious mononucleosis group, the serum ALT level was lowest and was significantly lower in comparison to the DILI, HBV, and HAV groups. (b) Serum T-bil levels according to the etiology of acute liver injury. The serum T-bil level differed significantly according to the etiology of acute liver injury (p<0.001). In the infectious mononucleosis group, the serum T-bil level was lowest and was significantly lower in comparison to the HBV and HAV groups. (c) The M2BPGi-to-ALT ratio according to the etiology of acute liver injury. This ratio differed significantly according to the etiology of acute liver injury (p<0.0001). In the infectious mononucleosis group, this ratio was highest and significantly higher in comparison to other etiologies, with the exception of autoimmune liver disease. (d) The M2BPGi-to-T-bil ratio according to the etiology of acute liver injury. This ratio differed significantly according to the etiology of acute liver injury (p<0.01). In the infectious mononucleosis group, this ratio was highest and was significantly higher in comparison to the autoimmune liver disease, HBV and HAV groups. Differences were evaluated by the Kruskal-Wallis test, and significant differences were verified by Dunn’s multiple comparison test. *p<0.05. AI: autoimmune liver disease, IM: infectious mononucleosis, DILI: drug-induced liver injury, HBV: hepatitis B virus, HAV: hepatitis A virus, EU: etiology unknown, ALT: alanine aminotransferase, M2BPGi: Mac-2-binding protein glycosylation isomer