Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2020 Jul 28;26(28):4094–4107. doi: 10.3748/wjg.v26.i28.4094

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

©The Author(s) 2020. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

PMC Copyright notice

Celecoxib inhibits apoptosis of hepatocytes and expression of caspase-3 and caspase-12 in thioacetamide-induced liver fibrosis. A: Immunohistochemical staining (100 ×) for collagen III, cyclooxygenase-2 (COX-2), caspase-12, and caspase-3 in the three groups of rats. Positive rates were analyzed using Image-Pro Plus 6.0. The data are expressed as the mean ± SD (n = 15, ^aP < 0.05 vs TAA + celecoxib group; ^bP < 0.01 vs control group). The scale bar is 200 μm; B: Western blot results showing collagen III, COX-2, caspase-3, and caspase-12 expression in the three groups. ^aP < 0.05 vs TAA + celecoxib group; ^bP < 0.01 vs control group. TAA: Thioacetamide; COX-2:Cyclooxygenase-2; GAPDH: Glyceraldehyde-3-phosphate dehydrogenase.

HHS Vulnerability Disclosure