Table 2.

Overview of organotypic slice culture studies which have used the prion-like features of aggregate-prone proteins from other neurodegenerative diseases.

Prion Feature	Amyloid Beta	Tau	α-Synuclein	TDP-43	SOD1	Huntingtin
Seeded aggregation	[36,40,41]	[49]	[63,64]		[71]
Prion-like spreading		[50,51]	[64,65]			[75]
Pathology induced by seeding	[36,47]		[63]	[70]		[75]
Strains	[36]		[66]		[71]
Drug screening (to prevent aggregation/spread)	[45,46]	[48]	[63]		[72]	[73,76,77]