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. 2020 Jun 28;12(7):1713. doi: 10.3390/cancers12071713
APE1i APE1 inhibitors
ATMi ATM inhibitors
ATRi ATR inhibitors
BER base excision repair
CHEK1i CHEK1 inhibitors
DDR DNA damage response
DSB double-strand break
EMA European Medicine Agency
EOC epithelial ovarian carcinoma
FDA Food and Drug Administration U.S. agency
GWAS genome wide association study
HGSOC high-grade serous ovarian carcinoma
HR homologous recombination repair
MMR mismatch repair
MSI microsatellite instability
NER nucleotide excision repair
NHEJ non-homologous end joining
OvC ovarian cancer
OS overall survival
PARPi poly(ADP-ribose) polymerase inhibitors
PFS progression-free survival
SNP single-nucleotide polymorphism
SSB single-strand break
WEE1i WEE1 inhibitors
wt wild-type