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. 2020 Jun 23;22(3):2052–2062. doi: 10.3892/mmr.2020.11266

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Statin rosuvastatin increases the viability of HCAECs induced by CoCl₂. (A) CoCl₂ decreased viability of HCAECs in a dose-dependent manner (0, 10, 20 and 30 mM) compared with the non-treated group. (B) CoCl2 decreased viability of HCAECs in a time-dependent manner (24, 48 and 72 h) compared with the non-treated group. (C) Rosuvastatin increased the viability of HCAECs in dose-dependent manner (0, 1.0, 2.0 and 3.0 mg/ml) compared with the non-treated group. (D) Statin increased viability of HCAECs in a time-dependent manner (24, 48 and 72 h) compared with non-treated group. *P<0.05, **P<0.01; ns, not significant; HCAECs, human coronary artery endothelial cells.