Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2020 Jun 23;22(3):2052–2062. doi: 10.3892/mmr.2020.11266

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

PMC Copyright notice

Figure 6. — In vivo efficacy of statin rosuvastatin on myocardial infarction. (A) Statin treatment markedly decreased the mitochondrial ROS in a myocardial infarction rat model. (B) Statin treatment decreased myocardial infarction area in the experimental rats. (magnification, ×50). (C) Statin treatment decreased thrombogenesis in the experimental rats after the 60-day treatment. (D) Statin treatment decreased Bax and Bad production in the experimental rats after the 60-day treatment. (E) Statin improved the cardiac function indicators LVPWd and LVEDD. (F) Statin increased expression level of JAK and STAT3 in myocardial tissue. Scale bar, 50 µm. *P<0.05, **P<0.01. ROS, reactive oxygen species; LVPWd, left ventricular end-diastolic posterior wall thickness; LVEDD, left ventricular end-diastolic diameter; JAK, Janus kinase; STAT, signal transducer and activator of transcription.