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. 2020 Aug 7;5:122. doi: 10.1038/s41392-020-0179-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Strategy for investigating the immunity-related effects of MET on liver cancer growth. b, c Impact of MET depletion on liver cancer growth in immunodeficient mice. NCG mice were individually transplanted s.c. with wild-type (WT) and MET-deficient (MET-KO) H22 cells (b) or Hepa1-6 cells (c), respectively. Tumor size was monitored at the indicated times. d, e Impact of MET depletion on liver cancer growth in the immunocompetent mice. C57BL/6 mice were individually transplanted s.c. with WT and MET-KO H22 cells (d) or Hepa1-6 cells (e), respectively. Tumor size was monitored at the indicated times. *p-Value < 0.05, **p-value < 0.01, ***p-value < 0.001, ns (nonsignificant), compared to the indicated groups; n = 10 per group.