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. 2020 Aug 7;10:13330. doi: 10.1038/s41598-020-70253-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Survival and toxicity mediated by kinetin within mammalian cellular systems. (A) Vitality assay for HL-60; (B) comet assay for HL-60; (C) vitality assay for HDF; (D) comet assay for HDF; (E) vitality assay for A549; (F) vitality assay for WI38. Cells treated with different concentrations of kinetin and/or 75 µM H₂O₂ *Significantly different from negative control and ^∆significantly different from H₂O₂ treated cells. Data are shown as averages ± SD from three independent experiments. Statistical significance among multiple groups was tested by the Kruskal–Wallis test. Individual groups were then tested using the Mann Whitney U-test and results were considered significant if the p-value was < 0.05.