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. 2020 Aug 3;10:1164. doi: 10.3389/fonc.2020.01164

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Copyright © 2020 Hoang, Matossian, Ucar, Elliott, La, Wright, Burks, Perles, Hossain, King, Browning, Bursavich, Fang, Del Valle, Bhatt, Cavanaugh, Flaherty, Anbalagan, Rowan, Bratton, Nephew, Miele, Collins-Burow, Martin and Burow.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Proposed mechanism of MEK5/ERK5 signaling in triple negative breast cancer. ERK5 mediates breast tumor formation through cell intrinsic and cell extrinsic mechanisms. Pro-angiogenic factors and ECM components converge in a signaling node that is regulated by ERK5 to influence ECM integrity and functionality. Schematic figure was created with BioRender.com.