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. 2020 Jul 2;68(6):1166–1170. doi: 10.1136/jim-2020-001284

Figure 4.

Figure 4

MiR-3150b-3p directly targeted TNFRSF11a in cervical cancer (CC) cells. The potential binding site of TNFRSF11a three prime untranslated region (3’-UTR) for miR-3150b-3p was predicted by bioinformatic analysis (TargetScan); luciferase activities in HEK293T cells co-transfected with wild-type (WT) 3’-UTR or mutated-type (MUT) 3’-UTR TNFRSF11a luciferase reporter plasmid and miR-3150b-3p mimics or mimic NC were determined by dual-luciferase reporter assay. (B–C) The mRNA and protein levels of TNFRSF11a in HeLa or SiHa cells with miR-3150b-3p mimic or inhibitor transfection. Data were shown as mean±SD. Each experiment was conducted independently for three times. **P<0.01, ***p<0.001 vs mimic/inhibitor NC group.