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. 2020 Aug 5;11:1191. doi: 10.3389/fphar.2020.01191

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Copyright © 2020 Long, Chen, Wu, Li, Gao, Huang, Zhang, Jia and Xia

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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FG-4592 attenuated DOX-induced mitochondrial oxidative stress with no effect on inflammation. (A) Serum MDA level of the experimental animals. (B) MDA content in the left ventricular from experimental animals. (C) Serum TNF-α level in the experimental animals. (D) Serum IL-6 level in the experimental animals. (E) TNF-α level in the left ventricular from experimental animals. (F) IL-6 level in the left ventricular from experimental animals (n = 7–9 per group). (G) mRNA levels of 13 mitochondria-encoded genes in the left ventricular tissues from experimental animals by qRT-PCR (n=6 per group). The values were represented as mean ± SED. *P < 0.05, **P < 0.01, and ***P < 0.001.