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. 2020 Aug 14;20:367. doi: 10.1186/s12872-020-01620-z

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Summary of significant Kaplan-Meier differences among Clusters. Kaplan-Meier estimates for the (1) time (years) from the initial diagnosis of subclinical diastolic dysfunction (DD) to HFpEF (left column), (2) age of HFpEF diagnosis, (3) time (years) from the development of HFpEF to death from all causes, and (4) age of death (right column). Each graph shows comparisons among the three Clusters A (black), B (red), and C (green). Kaplan-Meier analysis was performed on the (A) entire cohort, (B) female only cohort (C) male only cohort (D) all patients without left ventricular (LV) hypertrophy (E) all patients with LV hypertrophy (F) all patients who developed HFpEF and (G) all patients who remained with subclinical diastolic dysfunction (DD). P values = Cluster B versus Cluster C