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. 2020 Aug 14;11(8):632. doi: 10.1038/s41419-020-02902-2

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© The Author(s) 2020, corrected publication 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Transwell assays were conducted to observe the migratory cells in control and TGF-β, TGF-β + HHT, TGF-β + NVP-BHG712, or TGF-β + HHT + NVP-BHG712 treated HepG2 cells. Scale bars, 100 μm. b The migration rate of control and TGF-β, TGF-β + HHT, or TGF-β + HHT + EphB4 siRNA treated HepG2 cells observed through wound-healing assays. Scale bars, 50 μm. c Quantification of a (n = 5). d Quantification of b (n = 5). All data represent mean ± SEM. *p < 0.05, **p < 0.01, and ***p < 0.001; compared to the indicated groups.