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. 2020 Feb 3;69(2):309–323. doi: 10.1099/jmm.0.001162

Table 1.

E. coli strains used in this study

Strain

Genotype/serotype

Syndrome*

Spot test result†

Plaque formation‡

Source§

EN1E-0007

EAEC O86:H27

D

+

+

[66]

LRT9

EPEC O11:abH2

U

+

+

[67]

MC4100

F-araD139 Δ(argF-lac) U169 rpsL 150 StrR relA1 flhD5301 deoC1 ptsF25 rbsR

NP

+

+

[68]

BC/Ae017.2

EAEC

AMR, AMCR

D

UB||

BC/AaUIM099.2

EAEC

D

+

UB

BC/AaUIM171.3

EAEC

AMR

D

UB

BC/AaUIM206.1

EAEC

D

+

UB

BC/Ac277.1

EAEC

D

UB

BC/Ac338.3

EAEC

AMR, AMCR

D

+

+

UB

BC/AaUIM363.2

EAEC

D

+

+

UB

BC/Ac408.1

EAEC

D

+

UB

BC/Ac422.2

EAEC

D

UB

BC/Ac482.1

EAEC

AMR, SXTR

D

UB

BC/AaUIM517.1

EAEC

D

UB

BC/Ac512.2

EAEC

D

+

UB

*D, acute diarrhoea; U, details unknown; NP, non-pathogenic laboratory strain.

†For the spot test, phage in 5 µl drops were placed on streaks of the different clinical isolates, and then screened for clearing after overnight growth. A plus sign indicates that the strain was lysed by Escherichia virus PDX.

‡Plaque formation was assessed with serial dilutions of the PDX phage, propagated on different host strains using the pour plate method. In this case, individual plaques indicated reproductive infections [20]. A plus sign indicates PDX replicates (can form plaques on) the strain.

§Clinical isolate EN1E-0007 obtained from Tennessee, UB isolates obtained from Columbia

||Division of Pediatrics Infectious Diseases, University at Buffalo.