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. 2020 Aug 17;15:46. doi: 10.1186/s13024-020-00398-0

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — GluA4 levels in control, sporadic AD and DS CSF. a Box and whisker plots and individual points of the CSF levels of GluA4 (log2 intensity) grouped by clinical diagnosis. P-value resulting from one-way ANOVA on square root transformed GluA4 levels is shown in the top right corner. b CSF NPTX2 levels are plotted against CSF GluA4 levels with associated linear regression lines shown for controls, sporadic AD and DS groups (see legend)