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. 2020 Aug 3;11(8):646. doi: 10.1038/s41419-020-02808-z

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a The interaction between H3K27ac and PABPC1 promoter region was determined by ChIP assay. b The expression of SNHG14 in cytoplasm and nucleus of HCC cells. c The effect of SNHG14 on the direct binding between H3K27ac and PABPC1 promoter region was determined by ChIP assay. d Relative luciferase activity was determined by dual luciferase reporter assay. Renilla luciferase activity acted as an internal control. e, f PABPC1 mRNA ad protein levels were determined by qRT-PCR and western blotting. Data were representative images or were expressed as the mean ± SD of n = 3 experiments. *P < 0.05, **P < 0.01, ***P < 0.001.