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. 2020 Aug 3;11(8):646. doi: 10.1038/s41419-020-02808-z

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Photographs of tumor that developed in xenograft transplanted nude mouse tumor models in different groups. b Quantitative analysis of tumor volumes. c Quantitative analysis of tumor weights. d IHC analysis of Ki67 and CD31 in tumors of different groups. Scale bar, 50 μm. e Protein levels of the key components of PTEN signaling were determined by western blotting. β-actin served as a loading control. Data were representative images or were expressed as the mean ± SD of n = 3 experiments. *P < 0.05, **P < 0.01, ***P < 0.001.