Table 1:
Exposure–outcome associations with a probability of evidence for causation identified up to 2015
| Outcome | Strength of human evidence | Probability of causation, % | |
|---|---|---|---|
| Prenatal PBDEs | IQ loss and intellectual disability | Moderate to high | 70–100% |
| Prenatal organophosphate pesticides | IQ loss and intellectual disability | Moderate to high | 70–100% |
| Multiple prenatal exposures | Attention-deficit disorder | Low to moderate | 20–69% |
| Multiple prenatal exposures | Autism spectrum disorder | Low | 20–39% |
| Prenatal DDE | Childhood obesity | Moderate | 40–69% |
| Prenatal BPA | Childhood obesity | Very low to low | 20–69% |
| Adult DEHP | Adult obesity | Low | 40–69% |
| Adult DEHP | Adult diabetes | Low | 40–69% |
| Prenatal DDE | Adult diabetes | Low | 20–39% |
| Prenatal PBDEs | Cryptorchidism | Low | 40–69% |
| Prenatal PBDEs | Testicular cancer | Very low to low | 0–19% |
| Adult phthalates | Low testosterone, resulting in increased early mortality | Low | 40–69% |
| Adult benzyl and butyl phthalates | Male infertility, resulting in increased use of assisted reproductive technology | Low | 40–69% |
| Adult DEHP | Endometriosis | Low | 20–39% |
| Lifetime DDE | Fibroids | Low | 20–39% |