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. 2020 Aug 6;11:1258. doi: 10.3389/fimmu.2020.01258

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Copyright © 2020 Lee, Lee, Kashif, Yang, Nam, Song, Gong, Hong, Kim, Seok, Lee, Sung and Kim.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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S100A8 and S100A9 elicit apoptosis of eosinophils isolated from CEL and HES patients. (A–D) Eosinophils were isolated from CEL (n = 1) (A), HES (n = 2) (B), and reactive eosinophilia subjects (n = 5) (C), and polymorphonuclear cells and mononuclear cells were separated from AML subjects (n = 5) (D). Eosinophils from CEL, HES, and AML were incubated for 24, 48, or 72 h, and the cells from reactive eosinopohilia were incubated for 48 h with the indicated concentrations of S100A8 and S100A9. Apoptosis was analyzed by measuring the binding of annexin V-FITC and PI. Data are expressed as the means ± SD. *p < 0.05 indicates a significant difference between the control and stimulator-treated groups.