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. 2020 Jul 31;11:1150. doi: 10.3389/fphar.2020.01150

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Copyright © 2020 Li, Liu, Song, Zheng, Yi, Liu, Wang, Chu and Yang

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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H2S upregulates STZ-induced diabetic myocardial fibrosis mitophagy. (A) Representative images of transmission electron microscopy analysis the effect of STZ‐induced diabetic myocardial fibrosis in STZ-evoked mitophagy in the selected area. The insets in the above panels are magnified and are presented in the lower panel. Transmission electron micrographs at bars of 0.2 µm(Red arrow represents microscopy). (B) Western blotting analysis the expression of Atg5,Atg12,Atg16L1 in myocardial tissue treated with Control; STZ; STZ +H2S; H2S; n = 3, *p < 0.05 vs control. **p < 0.01 vs control. ^#p < 0.05 vs STZ. STZ, streptozotocin; H2S, hydrogen sulfide.