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. 2020 Aug 20;19:188. doi: 10.1186/s12944-020-01364-x

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Obesity modulates OXTR expression in epididymal fat in lean and obese mice. OXTR mRNA expression relative to a control gene, 18S rRNA (Panel a, n = 9 per group; p < 0.05 comparing lean and db/db mice), Panels b and c; Distribution of Oxtr and adiponectin (Adipoq) mRNA, expression in total tissue, adipocyte fraction and stromal vascular fraction (SVF). Data were expressed relative to mRNA level in the total tissue for each respective group (n = 4 for lean, n = 4 for db/db). Oxtr and Adipoq expression were significantly greater in in the adipocyte fraction compared to the SVF fraction (both p < 0.05). Western Blot of OXTR from adipose tissue membrane fractions (Panel d). Molecular weight in kilodaltons (kDa) is indicated on the left. Densitometric quantification of OXTR protein for 67 and 43 kDa bands (Panel e)

Data were expressed as mean ± SEM. * indicates p < 0.05.