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. 2020 Aug 22;11(8):698. doi: 10.1038/s41419-020-02912-0

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a The mRNA level of ELA in the renal medulla of DOCA/salt-treated rats (n = 6 per group). b Diagram of the animal protocol for ELA treatment in this study. c Representative immunohistochemical staining of ELA-histag in renal cortex and medulla. Kidneys without ELA-his tag transfection were used for Negative control. Scale bar, 100 µm. d The mRNA level of ELA in the kidneys of control group, DOCA/salt-treated group and DOCA/salt + ELA treated group (n = 6 per group). *P < 0.05 versus Control group; ^#P < 0.05 versus DOCA/salt group.