Table 4.
Snail survey results
| Number of snails | Schistosoma haematobium | Schistosoma bovis | S haematobium–S bovis (hybrid 1) | S haematobium–S bovis (hybrid 2) | S curassoni | S mansoni | Total infected snails (n [median Bayesian posterior prevalence estimate, 95% BCI]) | |
|---|---|---|---|---|---|---|---|---|
| Richard Toll and Lac de Guiers | ||||||||
| Bulinus truncatus and Bulinus globosus | 2532 | 60 (2·37%) | 15 (0·59%) | 25 (0·98%) | 6 (0·24%) | 0 | 0 | 88 (3·71, 2·98–4·55) |
| Biomphalaria pfeifferi | 407 | 0 | 0 | 0 | 0 | 0 | 9 (2·21%) | 9 (2·53, 1·25–4·49) |
| Barkedji and Linguère | ||||||||
| Bulinus umbilicatus | 4694 | 6 (0·13%) | 0 | 0 | 0 | 15 (0·32%) | 0 | 21 (0·49, 0·31–0·72) |
Summary of number (%) of Bulinus and Biomphalaria snails shedding each schistosome genotype over five malacological surveys. BCI=Bayesian credible interval. Hybrid 1 (where the S bovis cox1 mitochondrial DNA profile is associated with the S haematobium nuclear ITS profile) represents miracidia that are the product of repeated backcrossing of hybrids with S haematobium, resulting in biased homogenisation towards this species and ITS sequences that appear as just one species. Hybrid 2 miracidia exhibit either S bovis or S haematobium cox1 profiles associated with both S haematobium and S bovis parental nuclear ITS copies, appearing as double peaks on the four species-specific mutation sites on chromatograms.