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. 2020 Aug 24;11:364. doi: 10.1186/s13287-020-01877-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — MiR-617 directly targets Smad3. a The Venn plots showed predicted downstream genes binding miR-617. b, c Go annotation (b) and KEGG pathway analysis (c) showed the top 25 target genes and their enriched pathways. GO: A field directly related to reproduction. d Binding sequences in 3′UTR of miR-617 and Smad3 predicted online. The red letters represent the binding sequence of miR-617. Luciferase activity in 293T cells co-transfected with miR-617 mimics/NC and wild-type/mutant-type smad3, respectively. GO: Gene ontology; KEGG: Kyoto Encyclopedia of Genes and Genomes