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. 2020 Mar 25;123(5):1657–1670. doi: 10.1152/jn.00701.2019

Fig. 5.

Fig. 5.

Comparisons of zolmitriptan (Zol) effects on the firing properties of the bursting neurons following acute and chronic spinal transection. A–F: group data comparing absolute changes from controls of zolmitriptan effects (i.e., zolmitriptan minus control; mean marked by X) on the firing properties of the bursting neurons following acute and chronic spinal transection in response to increasing stimulus intensity. A: evoked spike count for acute (n = 24; except n = 25 at 10×) and chronic (n = 23 for all intensities) spinal transection. B: field potential for acute (n = 24; except n = 25 at 10×) and chronic (n = 23 for all intensities) spinal transection. C: field latency for acute (n = 16, 1×; n = 22, 2×; n = 23, 5×; n = 24, 10×) and chronic (n = 23 for all intensities) spinal transection. D: burst duration for acute (n = 24; except n = 25 at 10×) and chronic (n = 23 for all intensities) spinal transection. E: first-spike latency for acute (n = 16, 1×; n = 21, 2×; n = 22, 5×; n = 21, 10×) and chronic (n = 14, 1×; n = 20, 2×; n = 21, 5×; n = 20, 10×) spinal transection. F: spontaneous firing rate for acute (n = 24; except n = 25 at 10×) and chronic (n = 22 for all intensities) spinal transection. Significant difference (*P < 0.05; †P < 0.01; ‡P < 0.001) using a two-way unbalanced ANOVA is shown. The box and whisker plots show the minimum, first quartile, median, third quartile, and maximum, without outliers.