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The anti‐tumour effect of propofol by upregulation of miRNAs in cancer cells. AKT, protein kinase B; Bcl‐2, B‐cell lymphoma‐2; JAK, Janus kinase; miR, microRNAs; MMP, matrix metalloproteinase; NF‐κB, nuclear factor‐kappa B; STAT, signal transducer and activator of transcription 3; Wnt, wingless and proto‐oncogene integration‐1. “Arrows from propofol to → targets” means “activating targets.” “Blockade from propofol to targets” means “inhibiting targets”
