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. 2020 Aug 10;46(4):1477–1489. doi: 10.3892/ijmm.2020.4699

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Copyright: © Huang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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S100A1 mediates FOXD2-AS1-induced cell proliferation, migration and invasion in breast cancer. Following transfection with pcDNA-FOXD2-AS1, cells were transfected with shRNA-S100A1. (A and B) CCK-8 assay was performed to determine the proliferation of the differently treated cells. (C) Migratory ability of MCF-7 cells in the different treatment groups was determined by wound healing assay. (D) Invasive ability of MCF-7 cells in the different treatment groups was determined by Transwell assay. (E) Migratory ability of MDA-MB-468 cells in the different treatment groups was determined by wound healing assay. (F) Invasive ability of MDA-MB-468 cells in the different treatment groups was determined by Transwell assay. ^***P<0.001, vs. pcDNA-NC; ^##P<0.01 and ^###P<0.001, vs. FOXD2-AS1 + shRNA-NC. S100A1, S100 calcium binding protein A1.