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. 2020 Aug 7;44(4):1539–1549. doi: 10.3892/or.2020.7718

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Copyright: © He et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Inhibition of GLUD1 rescues the effect of miR-30a-5p inhibitor on oxidative stress in HK-2 renal tubular epithelial cells. (A) The protein expression of GLUD1 in HK-2 cells detected by western blot analysis. (B) The production of ROS in HK-2 cells induced by H/R. (C) The activity of SOD in HK-2 cells induced by H/R. (D) The activity of CAT in HK-2 cells induced by H/R. (E) The activity of GPx in HK-2 cells induced by H/R. *P<0.05 vs. H/R + NC2 + si-control, ^#P<0.05 vs. H/R + inhibitor + si-GLUD1.