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. 2020 Jul 28;22(4):2655–2664. doi: 10.3892/mmr.2020.11378

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Copyright: © Sun et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — TMP alleviates renal tubular pathological injury and improves renal function in rats with renal I/R. (A) Photomicrographs of periodic acid-Schiff staining showing typical renal tubular injury in Sham, I/R and I/R+TMP rats, red boxes highlight the corresponding areas of the high-power images. (B) Summarized data showing the renal tubular injury score in each group. (C) The ELISA results showing the level of KIM-1 in each group. (D and E) SCr and BUN levels in each group were measured by a biochemical analyzer. (F and G) The ELISA results showing the levels of the inflammatory cytokines TNF-α and IL-6 in each group. *P<0.05 vs. Sham rats; ^#P<0.05 vs. I/R rats. I/R, ischemia-reperfusion; TMP, tetramethylpyrazine; KIM-1, kidney injury molecule 1; TNF-α, tumor necrosis factor-α; IL-, interleukin; SCr, serum creatinine; BUN, blood urea nitrogen.