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. 2020 Jul 16;22(4):2665–2672. doi: 10.3892/mmr.2020.11345

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — LD delivery to lysosomes for degradation is impaired in HUVECs exposed to ox-LDL. HUVECs were untreated (basal) or treated with either 50 µg/ml LDL (control group) or 100 µg/ml ox-LDL for 6, 12, 24 or 48 h. Co-localization of LC3 (red) and LDs (green) in HUVECs following exposure to ox-LDL for (A) 6, (B) 12, (C) 24 and (D) 48 h. Co-localization of LAMP1 (red) and LDs (green) in HUVECs following exposure to ox-LDL for (E) 6, (F) 12, (G) 24 and (H) 48 h. Magnification, ×400. **P<0.01 (n=3). HUVEC, endothelial cell; ox-LDL, oxidized low-density lipoprotein; LD, lipid droplet; LAMP1, lysosomal-associated membrane protein 1.