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. 2020 Aug 28;3:477. doi: 10.1038/s42003-020-01210-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — LocusCompare plots³⁰ showing the relationship between PALMD eQTL and GWAS association with atrial fibrillation and stroke for variants located within 1 Mb of PALMD. a Esophagus muscularis and atrial fibrillation, colocalization PP4 = 97.0%. b Subcutaneous adipose tissue and cardio-embolic stroke, colocalization PP4 = 5.4%. c Aortic valve and all ischemic stroke, colocalization PP4 = 15.7%. GWAS associations were obtained from Nielsen et al.³¹ and the MEGASTROKE Consortium³². The lead GWAS variant is annotated.