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. 2020 Aug 28;11:4328. doi: 10.1038/s41467-020-18167-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Representative traces of Bay k8644-mediated potentiation of EFS-stimulated BSM contraction, summarized in d (n = 7 BSM strips). b Representative traces of Bay k8644-mediated potentiation of EFS-stimulated BSM contraction fully inhibited by ketamine and by nifedipine in dose-dependent manners, summarized in e (n = 8 BSM strips) and f (n = 12 BSM strips). c representative traces of ketamine-induced and nifedipine-induced inhibition of EFS-stimulated BSM contraction rescued by 50 or 200 nM Bay k8644, summarized in g (n = 17 BSM strips) and h (n = 13 BSM strips). Data are presented as mean values ± SD. These data support ketamine as an inhibitor of Cav1.2-mediated BSM contraction. Source data are provided as a Source Data file.