. Author manuscript; available in PMC: 2021 Jul 1.

Published in final edited form as: Eur J Endocrinol. 2020 Jul;183(1):63–71. doi: 10.1530/EJE-20-0129

Table 4.

Comparison between women with nonclassic 21-hydroxylase deficiency (N21OHD) and women with PCOS

	F N21OHD^* (N=22)	PCOS (N=27)	Ratio of medians N21OHD/Control	p
Age (years)	30 [23–36]	28 [22–35]		0.73
BMI (kg/m²)	27 [23–33]	31 [27–36]		0.12
A4 (nmol/L)	5.4 [3.8–8.3]	5.9 [4–9.9]	0.9	0.37
T (nmol/L)	1.4 [0.9–2.7]	2.1 [1–4.2]	0.7	0.24
11OHA4 (nmol/L)	13.2 [7.6–33.4]	4.4 [3.1–8.6]	3.0	<0.0001
11KA4 (nmol/L)	1.3 [0.7–1.8]	0.7 [0.6–0.9]	1.9	0.0054
11OHT (nmol/L)	1.3 [0.8–2]	0.5 [0.4–0.7]	2.8	<0.0001
11KT (nmol/L)	2.7 [1.2–3.8]	1.0 [0.6–1.3]	2.6	<0.0001

3 pre-pubertal girls were excluded from this group, to preserve comparable ages between the two groups.

Data are expressed as median [interquartile range]. A4, androstenedione; T, testosterone; 11OHA4, 11β-hydroxyandrostenedione; 11KA4, 11-ketoandrostenedione; 11OHT 11β-hydroxytestosterone; 11KT, 11-ketotestosterone; F, females.