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. 2020 Aug 14;21(16):5852. doi: 10.3390/ijms21165852

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Loss of PYR1 function provokes the setting of hallmarks characteristic of primed immunity. (A) Comparative RT-qPCR of SBT3.3, NRPD2, and NRPE1 transcript levels between healthy Col-0 and pyr1-2 plants. The expression was normalized to the constitutive ACT2/8 gene and then to the expression in Col-0 plants. (B) Chromatin immunoprecipitation (ChIP) and comparison between Col-0 and pyr1-2 plants of the level of histone H3 Lys4 trimethylation (H3K4me3) and histone H3 Lys9 acetylation (H3K9ac) on the SBT3.3, PR-1, WRKY6, and WRKY53 gene promoters as present in leaf samples. The setting of histone marks in ACTIN2 was used as an internal control. Data are standardized for Col-0 histone modification levels. Data represent the mean ± SD; n = 3 biological replicates. An ANOVA was conducted to assess significant differences between MPKs activation and PR1 accumulation (p < 0.05); the asterisks * above the bars indicate statistically significant differences regarding Col-0 plants.