Figure 1.

Autopsy kidneys deomstarted arteriosclerosis, variable degrees of autolysis and, rarely, fibrin thrombi. (A) A low-magnification view reveals complete autolysis in an autopsy with a prolonged PMI. Tubular nuclei are not visible, and tubular injury cannot be assessed. In contrast, chronic changes of glomerulosclerosis, arteriosclerosis, and TA/IF are still visualized. (Hematoxylin and eosin, ×100.) (B) A glomerulus exhibits mild changes of NDGS. (Hematoxylin and eosin, ×400.) (C) A glomerulus displays red blood cell congestion and an intracapillary fibrin thrombus. (Hematoxylin and eosin, ×400.) (D) A fibrin thrombus is seen in the lumen of an artery. (Hematoxylin and eosin, ×400.) (E) In this area of microscopic infarction, tubules exhibit coagulative-type necrosis, and there is prominent neutrophil infiltration with neutrophilic debris. (Hematoxylin and eosin, ×200.) (F) In this patient with hypertensive arterionephrosclerosis, an artery exhibits severe intimal sclerosis, compromising >50% of the lumen. (Hematoxylin and eosin, ×400.)