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. 2020 Sep 3;20:428. doi: 10.1186/s12935-020-01487-2

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — ANXA11 is involved in EIF3J-AS1-medited glioma cell functions. a The expression level of ANXA11 was evaluated by qRT-PCR after transfecting with pcDNA3.1/ANXA11. b, c Colony formation and EdU assays were combined to study the rescuing effect of pcDNA3.1/ANXA11 on sh-EIF3J-AS1 induced proliferation. d, e TUNEL and caspase 3/8/9 activity assays were utilized to study the rescuing effect of pcDNA3.1/ANXA11 on sh-EIF3J-AS1 induced apoptosis. **P < 0.01