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. 2020 Sep 3;148:46–49. doi: 10.1016/j.yjmcc.2020.08.017

Fig. 1.

Fig. 1

ACE2 expression in human and rat cardiac cell types and potential binding miRNAs (A) Relative expression of Ace2 in different mouse organ panels (N = 3 mice; FC: fold change); (B) Relative expression of ACE2 in HCF (human cardiac fibroblast), hiPSC-CM (human induced Pluripotent Stem Cell-derived Cardiomyocyte), HUVEC (Human Umbilical Vein Endothelial Cells) (N = 3, independent experiments, FC: fold change, control: hiPSC-CM); (C) Relative expression of Ace2 in NRCF (Neonatal Rat Cardiac Fibroblast) and NRCM (Neonatal Rat Cardiomyocytes) (N = 3, FC: fold change, control: NRCM); (D) Bioinformatics prediction of the potential binding sites of miR-200b, −200c and −429 in 3’UTR of Ace2.