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. 2020 Jul 28;4(9):1302–1315. doi: 10.1002/hep4.1558

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© 2020 The Authors. Hepatology Communications published by Wiley Periodicals LLC on behalf of American Association for the Study of Liver Diseases.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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FIG. 4 — CHS‐131 does not affect the fibrosis stage but reduces markers of fibrosis, stellate cell activation, and inflammation. Liver content in (A) hydroxyproline, (B) Col1a1, (C) α‐SMA, and (D) Gal‐3. Images below (B‐D) represent IHC from NASH+vehicle and NASH+CHS‐131 high dose. For P < 0.05 (one‐tailed ANOVA), *P < 0.05, **P < 0.01, ***P < 0.001 for post‐hoc LSD test for chow+vehicle (black stars) or NASH‐CHS‐131 low dose (red stars) or NASH‐CHS‐131 high dose (blue stars) compared to NASH+vehicle. Data represent means ± SEMs.