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. 2020 Aug 25;20(5):7. doi: 10.3892/etm.2020.9135

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Copyright: © Ding et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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UBR5 knockdown inhibits gastric cancer cell proliferation, invasion and migration. The transfection efficiency of UBR5 siRNAs was (A) determined by western blotting and (B) semi-quantified. (C) Cell proliferation was assessed by performing an MTS assay. Cell invasion was (D) assessed using a Transwell invasion assay (magnification, x200) and (E) quantified. (F) Cell migration was assessed by performing a wound healing assay. (G) Representative images of the wound healing assay (magnification, x200). *P<0.05 vs. control siRNA. UBR5, ubiquitin protein ligase E3 component N-recognin 5; siRNA, small interfering RNA.