Table 1.
Age, mean (±SD) | 68 (7) |
Sex, male, no. (%) | 16 (100) |
Race, white, no. (%) | 16 (100) |
Baseline pathology and treatment status | |
NDBE subjects, no. (%) | 9 (56) |
Short segment (≤3 cm) BE, no. (%) | 2 (13) |
LGD subjects, no. (%) | 4 (25) |
Ablative treatment-naïve subjects, no. (%) | 1 (6) |
Short segment BE, no. (%) | 1 (6) |
Treated subjects, no. (%) | 3 (19) |
Residual short segment BE, no. (%) | 3 (19)* |
HGD/IMC subjects, no. (%) | 3 (19) |
Ablative treatment-naïve subjects, no. (%) | 2 (13)† |
Short segment BE, no. (%) | 1 (6) |
Treated subjects, no. (%) | 1 (6) |
Residual short segment BE, no. (%) | 1 (6) |
Length of BE at study endoscopy, cm | |
Circumferential extent, mean (±SD) | 3.6 (4.3) |
Maximal extent, mean (±SD) | 5.1 (4.5) |
Short segment (≤3 cm) subjects, no. (%) | 8 (50) |
Long segment (>3 cm) subjects, no. (%) | 8 (50) |
Distance from diaphragmatic hiatus (D) to gastric folds (G), mean (±SD) | 2.3 (2.5) |
Subjects with sliding hiatal hernia (D-G >2 cm), no. (%) | 5 (31) |
Length of hiatal hernia, mean (±SD) | 5.6 (2.1) |
*One treated LGD patient had no visible BE on endoscopy and was classified as short segment BE.
†One HGD/IMC patient had prior endoscopic mucosal resection and no ablation, thus classified as ablative treatment-naïve.
BE, Barrett’s oesophagus; HGD, high-grade dysplasia; IMC, intramucosal carcinoma; LGD, low-grade dysplasia; NDBE, non-dysplastic BE.