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FAO inhibition prohibits GC cell growth. (A,B) Etomoxir treatment strikingly inhibited FAO in GC cells with ectopic CPT1C expression. (C,D) Etomoxir treatment strikingly inhibited cell viability in GC cells with ectopic CPT1C expression. (E,F) Etomoxir treatment strikingly induced cell cycle arrest in GC cells with ectopic CPT1C expression. *, P<0.05. FAO, fatty acid oxidation; GC, gastric cancer.
