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. 2020 Aug;8(15):923. doi: 10.21037/atm-20-1879

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2020 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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BK polyomavirus (BKPyV) infection in glomeruli. (A) The BKPyV-infected glomerular parietal epithelial cells presented with typical cytological changes in the peripheral tubular epithelium, with enlarged and ground-glass like nuclei and intra-nuclear inclusions (arrow) [hematoxylin-eosin (HE), ×400]. (B) Immunohistochemistry (IHC) staining showed SV40 large T antigen positivity in the BKPyV-infected glomerular parietal epithelial cells, and the contiguous proximal tubular epithelium (B, IHC, ×400). (C,D) BKPyV infection in both the visceral and parietal epithelium of Bowman’s capsule, with enlarged and hyperchromatic nuclei (C, HE, ×400) and SV40 large T antigen expression (D, IHC, ×400).