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. 2020 Aug;8(16):1007. doi: 10.21037/atm-20-5332

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2020 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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SNHG22/miR-429/SESN3 axis modulated the progression of ESCC cells. (A,B) The inhibition efficiency of miR-429 and overexpression efficiency of SESN3 was assessed by the qRT-PCR assay. (C) SESN3 expression was detected by qRT-PCR assay when SNHG22 was silenced, or miR-429 was overexpressed. (D,E) Rescue assay of CCK8 and colony formation assay were implemented to detect the proliferation. (F) Flow cytometry analysis rescue assay was implemented to detect the apoptosis of TE-10 and Eca109 cells. (G,H) Immunofluorescence assay and western blot assay were conducted to verify the autophagy of TE-10 and Eca109 cells. **, P<0.01. SNHG22, small nucleolar RNA host gene 22; ESCC, esophageal squamous cell carcinoma; qRT-PCR, quantitative real-time PCR.