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. 2020 Mar 29;11(5):1126–1136. doi: 10.1111/jdi.13235

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© 2020 The Authors. Journal of Diabetes Investigation published by Asian Association for the Study of Diabetes (AASD) and John Wiley & Sons Australia, Ltd

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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Activated autophagy can attenuate high glucose and aggravate hypoxia–reoxygenation (HR) injury in H9C2 cardiomyocytes by inhibiting nucleotide‐binding oligomerization domain‐like receptor protein 3 (NLRP3) inflammasome. (a) Western blot was used to analyze the expression of (b) LC3‐II/I, (c) p62, (d) NLRP3, (e) ASC, (f) caspase‐1 p20, (g) interleukin (IL)‐1β and (h) IL‐18. Data are shown as the mean ± standard error of the mean. *P < 0.05 versus normal glucose (NG, 5.5 mmol/L); ^& P < 0.05 versus high glucose (HG, 33 mmol/L); ^# P < 0.05 versus NG + HR; ^@ P < 0.05 versus HG + HR, P < 0.05; n = 5/group.